Oeste: Mal

It primarily circulates between wild birds and mosquitoes ( Culex tarsalis being a major vector). Horses and humans act as "dead-end" hosts, meaning they can contract the disease but typically do not develop high enough viral levels to pass it back to mosquitoes.

Hypersensitivity to sound/touch, wandering, and ataxia (lack of muscle coordination). mal oeste

Vision loss, circling, head pressing, and eventually paralysis or recumbency. Mortality rates in horses can range from 20% to 50% . Diagnosis and Management It primarily circulates between wild birds and mosquitoes

Outbreaks are often linked to wet seasons or heavy irrigation, which increase mosquito populations. Clinical Manifestations which increase mosquito populations.

Diagnosis is confirmed through (looking for IgM antibodies) or PCR to detect viral RNA in tissues or cerebrospinal fluid. Managing infectious equine neurologic disease (Proceedings)

The disease progresses through stages of neurological decline: Fever, depression, and lack of appetite.

The disease is caused by the Western Equine Encephalomyelitis Virus (WEEV) , a member of the Alphavirus genus in the Togaviridae family.